[{"command":"settings","settings":{"basePath":"\/","pathPrefix":"","setHasJsCookie":0,"ajaxPageState":{"theme":"maleinfertility","theme_token":"J5IUi6cGcXKkAfgotFxPk6fcykT2zRxFVhwPe3vD4_k"},"CToolsModal":{"modalSize":{"type":"scale","width":".9","height":".9","addWidth":0,"addHeight":0,"contentRight":25,"contentBottom":75},"modalOptions":{"opacity":".55","background-color":"#FFF"},"animationSpeed":"fast","modalTheme":"CToolsModalDialog","throbberTheme":"CToolsModalThrobber"},"panopoly_magic":{"pane_add_preview_mode":"automatic"},"wcm_analytics":"{\u0022token\u0022:\u0022032de332133100187eaeb103fa911fe0\u0022,\u0022proxy_server\u0022:\u0022https:\\\/\\\/mpp.sdsweb.weillcornell.org\u0022,\u0022cross_subdomain_cookie\u0022:false,\u0022debug_mode\u0022:false,\u0022ignore_dnt\u0022:false}","password":{"strengthTitle":"Password compliance:"},"type":"setting"},"merge":true},{"command":"insert","method":"replaceWith","selector":"div.pane-male-infertility-custom-panel-pane-5","data":"\u003Cdiv class=\u0022pane-male-infertility-custom-panel-pane-5\u0022\u003E\u003Carticle id=\u0022node-292\u0022 class=\u0022node node-glossary node-promoted clearfix\u0022\u003E\n\n  \n        \u003Ch3\u003EKartagener\u0026#039;s Syndrome\u003C\/h3\u003E\n    \n  \n  \u003Cdiv class=\u0022content\u0022\u003E\n    \u003Cdiv class=\u0022\u0022\u003E\n    \u003Cdiv class=\u0022field-content-items\u0022\u003E\n          \u003Cp\u003EA syndrome, first described in 1933 by Kartagener, which consisting of situs inversus, bronchiectasis and chronic sinusitis. Fifty percent of the patients with immotile cilia syndrome have features of Kartagener\u0027s Syndrome.\u003C\/p\u003E      \u003C\/div\u003E\n\u003C\/div\u003E\n  \u003C\/div\u003E\n\n  \n  \n\u003C\/article\u003E\n\u003Carticle id=\u0022node-291\u0022 class=\u0022node node-glossary node-promoted clearfix\u0022\u003E\n\n  \n        \u003Ch3\u003EKaryotype\u003C\/h3\u003E\n    \n  \n  \u003Cdiv class=\u0022content\u0022\u003E\n    \u003Cdiv class=\u0022\u0022\u003E\n    \u003Cdiv class=\u0022field-content-items\u0022\u003E\n          \u003Cp\u003EA chromosome analysis\u00a0\u003C\/p\u003E      \u003C\/div\u003E\n\u003C\/div\u003E\n  \u003C\/div\u003E\n\n  \n  \n\u003C\/article\u003E\n\u003Carticle id=\u0022node-293\u0022 class=\u0022node node-glossary node-promoted clearfix\u0022\u003E\n\n  \n        \u003Ch3\u003EKlinefelter\u0026#039;s Syndrome\u003C\/h3\u003E\n    \n  \n  \u003Cdiv class=\u0022content\u0022\u003E\n    \u003Cdiv class=\u0022\u0022\u003E\n    \u003Cdiv class=\u0022field-content-items\u0022\u003E\n          \u003Cp\u003EA chromosome abnormality with a 47 XXY pattern, which prevents normal male sexual development and causes reversible infertility due to the presence of an extra female (X) chromosome. These men present with azoospermia, gynecomastery and tall stature. Follicle stimulating hormone ( FSH) and luteinizing hormone ( LH) levels are increased, but testosterone ( T) decreased in 50% of cases. The testes are atrophic, firm and less than 2 cm. Testicular biopsy shows hyalinized tubules, relative Leydig cell hyperplasia and absence of sperm spermatogenesis.\u003C\/p\u003E      \u003C\/div\u003E\n\u003C\/div\u003E\n  \u003C\/div\u003E\n\n  \n  \n\u003C\/article\u003E\n\u003C\/div\u003E","settings":null}]